Hello everyone,
I am new to the forum. I am concerned about my bf's eating habits. High cholesterol and diabetes run in his family but he insists on eating foods that are mostly deep fried and fatty. I cannot get him to eat any fruits or vegetables unless I mix them in a meal like spaghetti or something. Does anyone have any ideas for how I can get him to start eating better foods? I've tried to talk to him but it's like he just doesn't want to face the facts. I am worried that he will end up killing himself with bad eating habits. Thanks for reading.
Full Version: High Cholesterol just a matter of time
Hi evmlrbw,
If you intend to engage in a Primate Breeding Program you have to cull him from the herd. In order to protect the survival of the fittest trait, you need to stop him from breeding and reducing the quality of the offspring. You have to select for proven evolutionary successful traits, or the maintenance costs escalate dramatically, and can wipe out the whole program.
Seriously, I can not understand how anyone can reach adulthood still clinging to these unhealthy practices, but to try to solve your dilemma you could try to encourage him to eat most of his meals from seafood, this will give him a lot more bang for his bucks in terms of nutrients and most carnivores like any type of meat. you could try cooking casseroles with lots of vegetables as well as lots of meat to make the vegetables taste like meat, you may be able to make pizza and smother the vegetables in cheese. What about baked vegetables covered in a meat based gravy.
I would try very hard to get him to understand the importance of good diet, research at the moment is propelling us towards personalised medicine, and they have recognised the importance of epigenetics and nutrigenomics, whereby what an ancestor has eaten may seriously change gene expression in turn causing disease in later offspring.
Good luck sounds like you are going to need it.
Issue 19 — May 27, 2009
Science News Roundup:
Chew Your Fruit: Before the Meal
Family Meals: Healthful Habits
Aerobic Fitness Protection Against Digestive Cancers
When Diet Meets Gene
The fact that our genes and diet interact is nothing new. Not everyone who eats a lot of junk food becomes overweight. Only some people taste the bitter compounds found in spinach. Then came the unraveling of the human genome, and the field of nutrigenomics soon followed. In nutrigenomics, scientists focus on how our genes influence the requirements and responses to nutrients.
In recent years, spurred by faster and cheaper DNA technologies, nutrigenomics has yielded novel insights into how our genes influence diet’s role in cancer and other chronic diseases. It is helping scientists understand how foods interact with genes and may someday help health practitioners identify who may gain the most cancer-fighting benefit from certain foods.
Finding the Link
“In nutrition, we’ve been treating everyone as if they’re the same, lumping them together, and then we get very noisy data,” says Steven H. Zeisel, MD, PhD, Director of the University of North Carolina’s Nutrition Research Institute and one of the panelists on AICR’s expert report. Imagine dealing with a population in which 40 percent of people have a gene variant that makes them respond to a nutrient. Depending on the mix of study participants, some might respond and others may not, “and so the study concludes it’s nonsense. But if we understood the importance of the gene variant you could predict who would respond. Now [that same study] makes tremendous sense and you can draw a conclusion.”
The majority of research in nutrigenomics involves single nucleotide polymorphisms (SNPs), referred to as “snips.” A SNP is a variant or misspelling in a single “letter” of our DNA (A, C, G, or T), a nucleotide.
Scientists have identified over 10 million SNPs thus far, with some common SNPs occurring in 5 to 50 percent of the population. It is estimated that every person carries approximately 50,000 such genetic misspellings. Many have no effect on gene expression. But a fraction of SNPs appear to play a role in a broad spectrum of cancer-related lifestyle factors, from fat metabolism to how our bodies interact with alcohol, folate, and vegetables.
Choline, found in eggs, meat, and wheat germ, is also needed for healthy fetal development.
SNPs at Work
Studies have found a number of relatively common SNPs influence nutrient requirements. In one of his recent studies, Dr. Zeisel and his colleagues identified a SNP that increases susceptibility to dietary deficiency of choline, an essential nutrient linked to influencing the risk of certain cancers, such as breast and colon. Three of four people in the UNC-area carry this gene variant, noted Dr. Zeisel.
Choline, found in eggs, meat, and wheat germ, is also needed for healthy fetal development. In England, scientists at the Institute of Food Research pinpointed a SNP found in half the population to explain why broccoli may decrease the risk of prostate cancer for some more than others.
Only the Beginning
Given the wealth of unexplored genes, experts say we are only in the early phase of understanding the diet-genotype connection to cancer. And while one SNP might influence how you react to a certain food, other foods may reduce the risk of cancer through different mechanisms.
“At the moment people are picking on the genes they think are important,“ says Richard Mithen, PhD, head of the Plant Natural Products and Health Programme at IFR, and lead author of the broccoli study. Dr. Mithen is beginning a 5-year follow-up study looking at hundreds of polymorphisms. “It’s finding the genes we don’t know about that’s next.”
The dietary advice to eat fruits and vegetables every day, and cut down on red meat and saturated fat is just really sound advice to everyone.” In the coming years, he adds, we hope to offer advice to fine-tune our diets to reduce the risk of progressive diseases like cancer. “But it’s still early.”
—Excerpted from a feature in ScienceNow.
Related Links:
AICR’s A Closer Look at Phytochemicals
Epigenetics: A New Frontier in Cancer Research
Human Genome Project: SNP Fact Sheet
Cheers
If you intend to engage in a Primate Breeding Program you have to cull him from the herd. In order to protect the survival of the fittest trait, you need to stop him from breeding and reducing the quality of the offspring. You have to select for proven evolutionary successful traits, or the maintenance costs escalate dramatically, and can wipe out the whole program.
Seriously, I can not understand how anyone can reach adulthood still clinging to these unhealthy practices, but to try to solve your dilemma you could try to encourage him to eat most of his meals from seafood, this will give him a lot more bang for his bucks in terms of nutrients and most carnivores like any type of meat. you could try cooking casseroles with lots of vegetables as well as lots of meat to make the vegetables taste like meat, you may be able to make pizza and smother the vegetables in cheese. What about baked vegetables covered in a meat based gravy.
I would try very hard to get him to understand the importance of good diet, research at the moment is propelling us towards personalised medicine, and they have recognised the importance of epigenetics and nutrigenomics, whereby what an ancestor has eaten may seriously change gene expression in turn causing disease in later offspring.
Good luck sounds like you are going to need it.
Issue 19 — May 27, 2009
Science News Roundup:
Chew Your Fruit: Before the Meal
Family Meals: Healthful Habits
Aerobic Fitness Protection Against Digestive Cancers
When Diet Meets Gene
The fact that our genes and diet interact is nothing new. Not everyone who eats a lot of junk food becomes overweight. Only some people taste the bitter compounds found in spinach. Then came the unraveling of the human genome, and the field of nutrigenomics soon followed. In nutrigenomics, scientists focus on how our genes influence the requirements and responses to nutrients.
In recent years, spurred by faster and cheaper DNA technologies, nutrigenomics has yielded novel insights into how our genes influence diet’s role in cancer and other chronic diseases. It is helping scientists understand how foods interact with genes and may someday help health practitioners identify who may gain the most cancer-fighting benefit from certain foods.
Finding the Link
“In nutrition, we’ve been treating everyone as if they’re the same, lumping them together, and then we get very noisy data,” says Steven H. Zeisel, MD, PhD, Director of the University of North Carolina’s Nutrition Research Institute and one of the panelists on AICR’s expert report. Imagine dealing with a population in which 40 percent of people have a gene variant that makes them respond to a nutrient. Depending on the mix of study participants, some might respond and others may not, “and so the study concludes it’s nonsense. But if we understood the importance of the gene variant you could predict who would respond. Now [that same study] makes tremendous sense and you can draw a conclusion.”
The majority of research in nutrigenomics involves single nucleotide polymorphisms (SNPs), referred to as “snips.” A SNP is a variant or misspelling in a single “letter” of our DNA (A, C, G, or T), a nucleotide.
Scientists have identified over 10 million SNPs thus far, with some common SNPs occurring in 5 to 50 percent of the population. It is estimated that every person carries approximately 50,000 such genetic misspellings. Many have no effect on gene expression. But a fraction of SNPs appear to play a role in a broad spectrum of cancer-related lifestyle factors, from fat metabolism to how our bodies interact with alcohol, folate, and vegetables.
Choline, found in eggs, meat, and wheat germ, is also needed for healthy fetal development.
SNPs at Work
Studies have found a number of relatively common SNPs influence nutrient requirements. In one of his recent studies, Dr. Zeisel and his colleagues identified a SNP that increases susceptibility to dietary deficiency of choline, an essential nutrient linked to influencing the risk of certain cancers, such as breast and colon. Three of four people in the UNC-area carry this gene variant, noted Dr. Zeisel.
Choline, found in eggs, meat, and wheat germ, is also needed for healthy fetal development. In England, scientists at the Institute of Food Research pinpointed a SNP found in half the population to explain why broccoli may decrease the risk of prostate cancer for some more than others.
Only the Beginning
Given the wealth of unexplored genes, experts say we are only in the early phase of understanding the diet-genotype connection to cancer. And while one SNP might influence how you react to a certain food, other foods may reduce the risk of cancer through different mechanisms.
“At the moment people are picking on the genes they think are important,“ says Richard Mithen, PhD, head of the Plant Natural Products and Health Programme at IFR, and lead author of the broccoli study. Dr. Mithen is beginning a 5-year follow-up study looking at hundreds of polymorphisms. “It’s finding the genes we don’t know about that’s next.”
The dietary advice to eat fruits and vegetables every day, and cut down on red meat and saturated fat is just really sound advice to everyone.” In the coming years, he adds, we hope to offer advice to fine-tune our diets to reduce the risk of progressive diseases like cancer. “But it’s still early.”
—Excerpted from a feature in ScienceNow.
Related Links:
AICR’s A Closer Look at Phytochemicals
Epigenetics: A New Frontier in Cancer Research
Human Genome Project: SNP Fact Sheet
Cheers
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